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- Disruption of Pseudomonas aeruginosa quorum sensing influences biofilm . . .
The opportunistic bacterial pathogen Pseudomonas aeruginosa is a leading cause of antimicrobial resistance-related deaths, and novel antimicrobial therapies are urgently required P aeruginosa infections are difficult to treat due to the bacterium's propensity to form biofilms, whereby cells aggregate to form a cooperative, protective structure Autolysis, the self-killing of bacterial cells
- Pseudomonas aeruginosa is capable of natural transformation in biofilms . . .
Pseudomonas aeruginosa is an opportunistic Gram-negative pathogen that produces large quantities of extracellular DNA (eDNA) that is required for biofilm formation P aeruginosa has a remarkable level of genome plasticity and diversity that suggests a high degree of horizontal gene transfer and recombination but is thought to be incapable of
- Comparison of Antibiofilm Activity of Pseudomonas aeruginosa Phages on . . .
Abstract The persistence of organisms as biofilms and the increase in antimicrobial resistance has raised the need for alternative strategies The study objective was to compare the ability of isolated bacteriophages to remove in vitro biofilms formed by Pseudomonas aeruginosa isolated from the environment with those isolated from diabetic and non-diabetic wounds
- Pseudomonas aeruginosa biofilm: treatment strategies to combat . . .
Pseudomonas aeruginosa is an opportunistic human pathogenic bacterium that is a common cause of both acute and chronic infections Multidrug-resistant P aeruginosa poses a significant challenge to antibiotics and therapeutic approaches due to its pathogenicity, virulence, and biofilm-forming ability mediated by quorum sensing Understanding the pathogenic mechanisms is essential for
- Pseudomonas aeruginosa biofilm infections: from molecular biofilm . . .
The present review is focussed on the important opportunistic pathogen and biofilm model organism Pseudomonas aeruginosa Initially, biofilm infections where P aeruginosa plays an important role are described Subsequently, current insights into the molecular mechanisms involved in P aeruginosa biofilm formation and the associated
- Optimization of an in vitro Pseudomonas aeruginosa Biofilm Model to . . .
Tobramycin and polymyxin B activity against biofilms Baseline PAO1 and AR0064 tobramycin MICs were 0 5 and 4 mg L, respectively, and polymyxin B MICs were both 0 5 mg L (Table 1) Tobramycin IV
- Ceragenin-mediated disruption of Pseudomonas aeruginosa biofilms
Introduction Biofilm formation is a colonization and survival strategy exploited by numerous microorganisms, such as Pseudomonas aeruginosa and other cystic fibrosis (CF) pathogens, often leading to chronic infections Biophysically, these microbial communities embedded in a matrix of autogenic extracellular polymers (EPS) are characterized by viscoelastic, i e , either solid (elastic) and
- Detection of biofilm formation among Pseudomonas aeruginosa isolated . . .
Pseudomonas aeruginosa is an aerobic, motile, nutritionally versatile, gram-negative, opportunistic pathogen It exhibits intrinsic resistance to many antibiotics Hence, infections caused by this bacterium are life-threatening such that patients may have longer hospital stay and even increased morbidity and mortality [5] For this bacterium, biofilm is also considered as an important virulence
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