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- Role of iron overload and ferroptosis in heart disease
<p indent="0mm">The essential trace element iron is a key component of hemoglobin, playing a role in erythropoiesis and oxygen transport In addition, iron plays a key role in several enzymatic and metabolic processes Iron deficiency causes anemia, whereas iron overload causes organ-related diseases such as heart failure, liver cirrhosis, and
- Role of ferroptosis in the pathogenesis of heart disease
In this review, we discuss molecular mechanisms that regulates ferroptosis in the heart, including pathways leading to iron overload and lipid peroxidation as well as the roles of key organelles in this process
- The molecular and metabolic landscape of iron and ferroptosis in . . .
In this Review, we summarize the relationship between the metabolic and molecular pathways of iron signalling and ferroptosis in the context of cardiovascular disease
- Role of iron overload and ferroptosis in heart disease (Cover Story)
In addition, we provide an overview of studies regarding the role that iron overload-associated cardiomyopathy and ferroptosis play in the pathogenesis and progression of heart disease
- p53 mediates iron overload and ferroptosis via transcriptional . . .
This effect is largely attributed to an increase in TfR1 expression in cardiomyocytes following PM2 5 exposure, which leads to excessive iron influx into the cells, resulting in iron overload and subsequent disruption of iron homeostasis
- The role of iron overload and ferroptosis in arrhythmia pathogenesis
We highlight the key molecular mechanisms governing iron overload and ferroptosis, and discuss potential molecular pathways linking ferroptosis with arrhythmias
- The Role of Macrophage Iron Overload and Ferroptosis in . . . - MDPI
Ferroptosis is a new type of cell death caused by iron-dependent lipid peroxidation In recent years, it has been found that ferroptosis can promote the progression of atherosclerosis (AS)
- The role of iron overload and ferroptosis in arrhythmia pathogenesis
We highlight the key molecular mechanisms governing iron overload and ferroptosis, and discuss potential molecular pathways linking ferroptosis with arrhythmias
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